Dehydrogenase

The University of Leeds researchers said the compounds work by preventing the enzyme dihydroorotate dehydrogenase, which is essential to the growth of the parasite Plasmodium falciparum, from working. ❋ Unknown (2009)

These steps are catalysed be the enzymes alcohol dehydrogenase and aldehyde dehydrogenase respectively. ❋ Unknown (2010)

Molecular basis and characterization of the hyperinsulinism/hyperammonemia syndrome: predominance of mutations in exons 11 and 12 of the glutamate dehydrogenase gene. ❋ Unknown (2010)

Stanley CA, Lieu YK, Hsu BY, Burlina AB, Greenberg CR, Hopwood NJ, et al. Hyperinsulinism and hyperammonemia in infants with regulatory mutations of the glutamate dehydrogenase gene. ❋ Unknown (2010)

The region includes genes encoding a CoA-dependent methylmalonate semialdehyde dehydrogenase (dntE), a putative NADH-dependent dehydrogenase (ORF13), and a bifunctional isomerase/hydrolase (dntG). ❋ Unknown (2010)

First, a few basics: the ailment is known as G6PD deficiency (for glucose-6-phosphate dehydrogenase) and, according to a 2008 article in The Lancet, it "is the most common human enzyme defect, being present in more than 400 million people worldwide." ❋ Randall Amster (2010)

As a result, most patients with glutamate dehydrogenase hyperinsulinism will be responsive to Diazoxide. ❋ Unknown (2010)

Deficiencies of pyruvate dehydrogenase (PDH) and pyruvate carboxylase (PDH and PC); Leigh syndrome ❋ Unknown (2010)

Other blood tests (your doctor will determine how often), including chemistries; lactate dehydrogenase; and adrenal hormones such as cortisol, testosterone, 17-hydroxyprogesterone, androstenedione and dehydroepiandrosterone ❋ Unknown (2009)

It has been observed in patients with2,5 and without4,7 glucose-6-phosphate dehydrogenase (G6PD) deficiency and is characterized by the presence of Heinz bodies. ❋ Unknown (2010)

First, a few basics: the ailment is known as G6PD deficiency for glucose-6-phosphate dehydrogenase and, according to a 2008 article in The Lancet, it "is the most common human enzyme defect, being present in more than 400 million people worldwide." ❋ Randall Amster (2010)

"Effects of H2-blockers on alcohol dehydrogenase ADH activity" ❋ M.D. Leo Galland (2010)

Because the gene for the Glucose – 6-posphate dehydrogenase (G6PD) enzyme is located on the X chromosome, the trait is far more common in men than in women and it is also disproportionately represented in certain communities. ❋ Unknown (2009)

I've never been a big drinker, never been in a drunken bar fight, never needed to go to rehab - like a lot of yellow people, I have one brave molecule of alcohol dehydrogenase somewhere in my blood stream fighting off that one pina colada I can drink before I call it a night. ❋ Frankwu (2007)

Probably on Friday I had a little too much cream pie and not enough lactase, a little too much red wine and not enough alcohol dehydrogenase, a little too much Chinese food and not enough rice, a little too much fried egg and not enough bread. ❋ Frankwu (2007)

Alcohol dehydrogenase (ADH) is one of the major enzymes involved in alcohol metabolism. ❋ Unknown (2007)

A form of the alcohol dehydrogenase gene may protect Afro-Trinidadians from developing alcoholism ❋ Unknown (2007)

But fava beans can cause hemolytic anemia -- an abnormal breakdown of red blood cells -- in people with an enzyme deficiency called Glucose-6-phosphate dehydrogenase (G6PD) deficiency. ❋ Unknown (2009)