In contrast, inhibitory cells called interneurons put the brakes on unbridled activity to maintain order and control. ❋ Unknown (2010)
If they wanted channelrhodopsin to work only in, say, parvalbumin-positive fast-spiking interneurons, they could use the promoter for that cell type. ❋ Michael Chorost (2011)
But they were wrong; one of the very first questions was: “What is the function of interneurons?” ❋ Unknown (2010)
The pair discovered that short, locally acting neurons called fast-spiking interneurons generate gamma waves, and that they enhance the information flow in long, distantly acting neurons. ❋ William Harryman (2009)
In both experiments, selectively stimulating the fast-spiking interneurons evoked gamma oscillations, thereby confirming the hypothesis that these neurons drive the gamma rhythm. ❋ William Harryman (2009)
The experimenters utilized optogenetics, developing custom-designed viruses to infect only the fast-spiking interneurons of either the prefrontal or barrel cortex in mice with genetically engineered, light-sensitive cation channels. ❋ William Harryman (2009)
The strongest hypothesis for the cellular mechanisms underlying generation of the gamma rhythm is that it is due to the activation of fast-spiking interneurons in the cerebral cortex. ❋ William Harryman (2009)
The interneurons then transform the electrical signals into a language that can be utilized as memory in other parts of the brain. ❋ Unknown (2009)
Then, they inserted fine optical fibers into the relevant region of the cortex, allowing light to be delivered to the infected neurons and thereby activating only the fast-spiking interneurons. ❋ William Harryman (2009)
The aversive tail stimuli that produced sensitization of the gill-withdrawal reflex activated modulatory interneurons that acted on terminals of the sensory neurons. ❋ Unknown (2001)
Next, we found that the sensory neurons made direct connections to the motor neurons as well as indirect connections through interneurons, both excitatory and inhibitory. ❋ Unknown (2001)
The serotonergic and other modulatory interneurons all acted on the sensory neurons and on their presynaptic terminals to enhance transmitter release from their presynaptic terminals. ❋ Unknown (2001)
Later, Hawkins and I found that tail stimuli that initiate sensitization activate a set of modulatory interneurons, the most important of which are serotonergic. ❋ Unknown (2001)
He also described two fundamental types of nerve cells, still named after him as neurons 'Golgi type I', extending their axons at a distance from the cell body (the 'projection neurons' of the modern nomenclature), and 'Golgi type II', with axons ramifying in the vicinity of the cell body (corresponding to the 'local circuit neurons' and 'interneurons' of the modern nomenclature). ❋ Unknown (1998)
The main cell types of the cerebral cortex i.e. small and large pyramidal neurons (A, B, C, D, E) and non pyramidal (F, K) cells (interneurons in the modern nomenclature) are superbly outlined. ❋ Unknown (1998)
Furthermore, the animals showed signs of a deficit of interneurons, nerve cells that connect other neurons in neural pathways. ❋ Unknown (2010)
Whether a similar signalling between a subpopulation of GABA-releasing astrocytes and interneurons may be involved in ictal discharge initiation in the EC represents an interesting question to be addressed in future studies. ❋ Marta Gómez-Gonzalo Et Al. (2010)
Given that inhibitory interneurons have been reported to restrain the recruitment of neurons during the development of the ictal discharge ❋ Marta Gómez-Gonzalo Et Al. (2010)
