Gago, N, et al. Progesterone and the oligodendroglial lineage: stage-dependent biosynthesis and metabolism. ❋ T.S. Wiley (2010)
In glial tumors, the loss of heterozygosity of the 1p and 19q chromosomal arms is thought to be a marker of good prognosis in oligodendroglial tumors. ❋ Karine S Durand (2010)
No co-localization of EYFP-expression and the oligodendroglial marker CC1 could be found in the cerebellar white matter (M-O) or the corpus callosum (P-R). ❋ Unknown (2009)
Slices were counterstained with the oligodendroglial marker CC1 (D). ❋ Unknown (2009)
In addition, the recombination observed in astrocytes (also in cerebellar Bergmann glia and retinal Müller cells) hints towards a role of oligodendroglial genes (PLP) in early astroglia differentiation, supporting the idea of PLP+ glia progenitors, which give rise to astrocytes ❋ Unknown (2009)
B: Engrafted hCNS-SCns migrated away from the injury and appeared to differentiate in a site-specific manner with oligodendroglial and neuronal morphologies in the white and grey matters, respectively. ❋ Mitra J. Hooshmand Et Al. (2009)
Manipulation of the individual Rho GTPases showed differential effects on survival, with little variation in their migratory route and predominant differentiation into the oligodendroglial lineage. ❋ Fujiki Numano (2009)
The resulting cell line (T-IhNSC) has the capacity to generate unlimited quantities of conditionally immortalized hNSCs, which can then be differentiated into mature neurons, astroglia and significant numbers of oligodendroglial cells. ❋ Unknown (2008)
This makes T-IhNSC the cell line with the highest oligodendroglial developmental potential to become available, to date. ❋ Unknown (2008)
In this context, we recently reported the establishment of v-myc, conditionally immortalized, human neural stem cell lines, which retain strict mitogen dependence in culture and generate mature neuronal and glial progeny including, for the first time, a significant percentage of oligodendroglial cells. ❋ Unknown (2008)
The oligodendroglial cells were labelled with GalC. ❋ Unknown (2008)
The current study focused on the effect of eIF2B5 mutation on the ER-stress response of an oligodendroglial-derived cell line. ❋ Liraz Kantor Et Al. (2008)
The data provide evidence that oligodendroglial-derived cells expressing a mutated eIF2B constantly use their stress response mechanism as an adaptation mean in order to survive. ❋ Liraz Kantor Et Al. (2008)
Since brain cells from affected individuals are not available for research, we wished to assess the effect of eIF2B mutation on oligodendroglial-derived cells. ❋ Liraz Kantor Et Al. (2008)
The current study provides evidence that oligodendroglial-derived cells are forced to elicit their adaptation capacity in order to survive in the face of a mutation in eIF2B5, since even under normal conditions they express high basal levels of ATF4, GADD34 and ER-associated Bip, PDIA1 and PDIA3. ❋ Liraz Kantor Et Al. (2008)
[94], suggesting neuronal damage, oligodendroglial loss, and subsequent demyelination post-trauma. ❋ Mitra J. Hooshmand Et Al. (2009)
Engrafted hCNS-SCns migrated along the spinal cord and appeared to differentiate in a site-specific manner consistent with neuronal and oligodendroglial morphologies in the grey and white matter, respectively ( ❋ Mitra J. Hooshmand Et Al. (2009)
A) Quantitative analysis of the expression of the oligodendroglial markers GalC. ❋ Unknown (2008)
